Follistatin 344

Follistatin-344 is a synthetic version of the naturally occurring Follistatin protein, which is encoded by the FST gene in humans.

It is an autocrine glycoprotein expressed in nearly all tissues of higher animals. Its primary function is to bind and neutralize members of the TGF-β superfamily, particularly activin, a paracrine hormone. Follistatin-344 is known for its role in inhibiting myostatin, a protein that limits muscle growth, leading to increased muscle mass in animal studies.

It also plays a role in cellular differentiation, tissue repair, and inflammatory responses. Research suggests potential applications in muscle growth, cancer treatment, liver protection, and diabetes management.

The above information is displayed for information purpose only, and has not been reviewed by EON nor does EON attests or validates the accuracy nor does it constitutes a recommendation or validation.

Follistatin-344 is a synthetic version of the naturally occurring Follistatin protein, which is encoded by the FST gene in humans.

It is an autocrine glycoprotein expressed in nearly all tissues of higher animals. Its primary function is to bind and neutralize members of the TGF-β superfamily, particularly activin, a paracrine hormone. Follistatin-344 is known for its role in inhibiting myostatin, a protein that limits muscle growth, leading to increased muscle mass in animal studies.

It also plays a role in cellular differentiation, tissue repair, and inflammatory responses. Research suggests potential applications in muscle growth, cancer treatment, liver protection, and diabetes management.

The above informationis displayed for information purpose only, and has not been reviewed by EON nor does EON attests or validates the accuracy nor does it constitutes a recommendation or validation.
Sources:
https://en.wikipedia.org/wiki/Follistatin
https://www.peptidesciences.com/follistatin-344-1mg
https://timesofindia.indiatimes.com/business/india-business/understanding-follistatin-344-peptide-muscle-research/articleshow/108243242.cms
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